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Thailand Third-Generation IVF PGT Technology: Disease Screening Scope and Limitations

Thailand third-generation IVF (PGT) can screen for chromosomal aneuploidy, structural abnormalities, and specific single-gene diseases. This article details the detection scope, indications, limitations, and clinical considerations of PGT-A, PGT-M, and PGT-SR by a reproductive doctor.

What diseases can Thailand third-generation IVF screen for?

Thailand's third-generation IVF technology, Preimplantation Genetic Testing (PGT), can analyze the chromosome number, structure, and specific single-gene pathogenic sites of embryos before transfer. Clinically, it is divided into three types based on testing purpose: PGT-A (aneuploidy screening), PGT-M (monogenic disease testing), and PGT-SR (chromosomal structural rearrangement testing). There are clear differences in the disease coverage, detection accuracy, and applicable populations for each technology.

I. Chromosomal abnormalities detectable by PGT-A

PGT-A (formerly PGS) is used to screen embryos for numerical chromosomal abnormalities (aneuploidy) and is currently the most widely used PGT technology. The following chromosomal abnormalities can be detected by PGT-A:

Abnormality TypeSpecific Disease / SyndromeChromosome Karyotype
Autosomal TrisomyDown Syndrome47,XX,+21 or 47,XY,+21
Autosomal TrisomyEdwards Syndrome47,XX,+18 or 47,XY,+18
Autosomal TrisomyPatau Syndrome47,XX,+13 or 47,XY,+13
Sex Chromosome AbnormalityTurner Syndrome45,X
Sex Chromosome AbnormalityKlinefelter Syndrome47,XXY
Sex Chromosome AbnormalityTriple X Syndrome47,XXX
Sex Chromosome AbnormalityXYY Syndrome47,XYY
Chromosomal Microdeletion22q11.2 Deletion Syndrome (DiGeorge Syndrome)del(22)(q11.2)
Chromosomal Microdeletion1p36 Deletion Syndromedel(1)(p36)
Chromosomal MicrodeletionAngelman Syndrome (some types)del(15)(q11-q13) maternal

The detection resolution of PGT-A is typically 5–10 Mb, with some platforms able to detect microdeletions larger than 1 Mb. Structural abnormalities such as balanced translocations and inversions require screening via PGT-SR.

II. Monogenic diseases detectable by PGT-M

PGT-M (formerly PGD) targets genetic diseases with known causative genes. It requires identifying the pathogenic mutation carried by one or both partners, followed by customized probe development. The following monogenic diseases have established PGT-M testing protocols clinically:

Disease CategorySpecific DiseaseCausative Gene
HemoglobinopathyAlpha-thalassemia, Beta-thalassemiaHBA1/HBA2, HBB
HemoglobinopathySickle Cell DiseaseHBB
Neuromuscular DiseaseSpinal Muscular Atrophy (SMA)SMN1
Neuromuscular DiseaseDuchenne Muscular Dystrophy (DMD)DMD
Neuromuscular DiseaseHuntington's DiseaseHTT
Hereditary Cancer SyndromeHereditary Breast and Ovarian Cancer (HBOC)BRCA1, BRCA2
Hereditary Cancer SyndromeLynch SyndromeMLH1, MSH2, MSH6, PMS2
Hereditary Cancer SyndromeFamilial Adenomatous Polyposis (FAP)APC
Coagulation DisorderHemophilia A, Hemophilia BF8, F9
Connective Tissue DiseaseMarfan SyndromeFBN1
Skeletal DysplasiaOsteogenesis Imperfecta (some types)COL1A1, COL1A2
Hereditary Hearing LossAutosomal Recessive Hearing LossGJB2, SLC26A4, etc.
Metabolic DiseasePhenylketonuria (PKU)PAH
Metabolic DiseaseGaucher DiseaseGBA

The detection scope of PGT-M depends on the clearly identified pathogenic mutation within the family. It cannot screen for genetic diseases with unknown causative genes, nor can it screen for polygenic diseases (such as type 2 diabetes, essential hypertension, schizophrenia, etc.).

III. Chromosomal structural abnormalities detectable by PGT-SR

PGT-SR is used to detect whether embryos carry chromosomal structural rearrangements, including reciprocal translocations, Robertsonian translocations, inversions, and insertions. Such abnormalities are common in patients with recurrent miscarriage, infertility, or oligoasthenozoospermia.

  • Reciprocal Translocation: Exchange of segments between two non-homologous chromosomes, can lead to unbalanced embryos
  • Robertsonian Translocation: Fusion of two acrocentric chromosomes at the centromere, commonly involving chromosomes 13, 14, 15, 21, 22
  • Pericentric / Paracentric Inversion: Inversion of a segment within a single chromosome, can produce unbalanced gametes during meiosis
  • Insertion: A segment from one chromosome inserted into another, relatively rare

PGT-SR is typically performed using SNP arrays or next-generation sequencing to distinguish between balanced and unbalanced embryos, helping carriers select chromosomally normal embryos for transfer.

How clinicians view the indications for PGT

From a reproductive medicine perspective, PGT is not a routine option for all IVF patients. In clinical decision-making, we primarily recommend PGT based on the following indications:

  • Female age ≥ 38 years (significantly increased risk of embryonic aneuploidy)
  • Recurrent pregnancy loss (≥ 2 miscarriages, excluding uterine anatomical and endocrine factors)
  • Previous pregnancy with chromosomal abnormality
  • One partner is a carrier of chromosomal structural rearrangement
  • Family history of known monogenic genetic disease
  • Repeated implantation failure (some studies suggest PGT-A may improve live birth rates)

It is important to clarify that PGT cannot improve embryo quality nor increase the number of available embryos. It only helps select chromosomally normal embryos from the existing pool. For patients with diminished ovarian reserve (low AMH, reduced antral follicle count), the number of obtainable embryos is limited, and the value of PGT needs individualized assessment.

Differences in PGT policies between Thailand and mainland China

Thailand's clinical application of PGT technology is relatively open, mainly reflected in the following aspects:

ItemThailandMainland China
PGT-A ApplicationApplicable for advanced age, recurrent miscarriage, repeated implantation failure, etc.Must meet medical indications and require ethical approval
PGT-M Disease ScopeCovers most monogenic diseases, including some tumor susceptibility genesLimited to severe genetic diseases, requires approval
Sex SelectionPermitted (under certain legal frameworks)Non-medical sex selection prohibited
Embryo Biopsy TimingUsually blastocyst biopsy on days 5–6Blastocyst biopsy, must comply with national standards
Genetic Testing PlatformNext-generation sequencing (NGS), SNP arrays, qPCRNGS, SNP arrays, FISH, etc.

Before undergoing PGT in Thailand, patients still need genetic counseling, informed consent, and necessary ethical review. For PGT-M, genetic test reports of both partners and family history of genetic diseases must be provided. Testing platforms and data analysis capabilities vary among different reproductive centers, directly affecting the range and accuracy of detectable diseases.

Easily overlooked testing limitations

Mosaicism Issue: Embryos may contain both normal and abnormal cell lines (mosaicism). Biopsy of 5–8 trophectoderm cells may not represent the entire embryo's chromosomal status. When the mosaic ratio is below 30%, routine PGT-A may misdiagnose it as normal.
Technical Testing Errors: The false positive rate for NGS-based PGT-A is approximately 1–3%, and the false negative rate is about 0.5–1.5%. The accuracy of monogenic disease testing depends on probe design and the quality of family linkage analysis.
Inability to Screen Polygenic Diseases: Diseases such as diabetes, hypertension, autism, and schizophrenia, which are determined by multiple genes and environmental factors, cannot currently be screened by PGT.
Non-genetic Congenital Malformations: Neural tube defects, cleft lip/palate, congenital heart disease, etc., are mostly caused by non-genetic factors and cannot be predicted by PGT.

Furthermore, PGT cannot replace prenatal diagnosis. After pregnancy, amniocentesis or chorionic villus sampling for chromosomal karyotype analysis or genetic testing is still recommended to confirm the actual fetal condition.

Common cognitive pitfalls patients fall into

Mistake 1: "PGT can screen for all genetic diseases." — In reality, PGT-M only targets monogenic diseases with known pathogenic mutations and requires customized testing protocols; it cannot cover all genetic diseases.
Mistake 2: "Embryos with normal PGT-A results guarantee 100% pregnancy success." — Normal embryonic chromosomes are a necessary condition for successful pregnancy, but not sufficient. Endometrial receptivity, maternal immune factors, and embryonic metabolism all affect the final outcome.
Mistake 3: "PGT can improve egg quality." — PGT is only used for embryo selection and does not improve egg or embryo quality. Egg quality is determined by age, ovarian reserve, lifestyle factors, etc.
Mistake 4: "PGT in Thailand can screen for all diseases without restrictions." — Thailand has clear regulations on the scope of PGT application, especially for PGT-M, which requires detailed genetic data submission; not all diseases are permitted for testing.

Actual testing process and timeline

From ovarian stimulation to obtaining PGT results, it typically takes 4–6 weeks. The specific steps are as follows:

  1. Ovarian Stimulation and Egg Retrieval (about 10–14 days): Use gonadotropins for controlled ovarian stimulation, followed by transvaginal ultrasound-guided egg retrieval.
  2. In Vitro Fertilization and Embryo Culture (5–6 days after egg retrieval): Conventional IVF or ICSI insemination, culture to blastocyst stage.
  3. Trophectoderm Biopsy (Days 5–6): Use a laser to create an opening in the zona pellucida of the blastocyst and aspirate 5–8 trophectoderm cells.
  4. Embryo Freezing (after biopsy): All biopsied blastocysts are immediately vitrified and stored while awaiting test results.
  5. Genetic Testing (7–14 days): Perform NGS, SNP array, or qPCR analysis based on the test type.
  6. Result Interpretation and Transfer (after testing): Select embryos that are chromosomally normal or do not carry the pathogenic mutation for frozen embryo transfer.

Throughout the cycle, genetic counseling should be completed before ovarian stimulation. For PGT-M testing, family samples need to be submitted 1–2 months in advance for probe design and validation.

Frequently asked questions

Q1: Can miscarriage still occur after transferring a PGT-normal embryo?
Yes. The miscarriage rate after transferring a PGT-normal embryo is about 5–10%, lower than the unscreened group, but the risk remains. Causes include non-chromosomal embryonic factors, maternal uterine environment, endocrine abnormalities, and immune factors.
Q2: What documents are needed for PGT in Thailand?
You need ID cards and passports (valid for more than 6 months) of both partners, notarized marriage certificate, chromosome karyotype analysis reports of both partners, genetic test reports related to genetic diseases (if any), and records of previous fertility and miscarriage history. Some reproductive centers also require infectious disease screening reports for HIV, syphilis, hepatitis B, and hepatitis C.
Q3: Can I still do PGT if my AMH is low?
Low AMH indicates diminished ovarian reserve, and the number of eggs retrieved and embryos available for testing may be limited. However, as long as there are enough blastocysts for biopsy, PGT can still be performed. A realistic assessment of the expected number of embryos and a discussion with your doctor about whether PGT is worthwhile are necessary.
Q4: How long does PGT testing take? Will it delay the transfer?
It usually takes 7–14 days from biopsy to obtaining the test report. The embryos remain frozen throughout the process, which does not affect their quality. Pregnancy outcomes with frozen embryo transfer are not significantly different from fresh embryo transfer.

Practitioner observations: Real clinical experience

In daily work at the reproductive center, I have observed the following noteworthy situations:

  • Some patients have overly high expectations of PGT, believing it guarantees a healthy child. It is necessary to fully inform them of the testing scope and technical limitations.
  • For carriers of chromosomal structural abnormalities (e.g., balanced translocations), the rate of identifying normal embryos through PGT-SR is typically only 20–35%, requiring mental preparation for multiple stimulation cycles.
  • Although the aneuploidy rate in advanced-age patients (≥40 years) is as high as 60–80%, PGT-A can still effectively screen for transferable embryos if a sufficient number of blastocysts are obtained.
  • In PGT-M testing, the availability of family samples directly affects the feasibility of the testing protocol. Some genetic diseases require cooperation from multiple family members, including parents and the proband.
  • The qualifications of genetics laboratories vary significantly among different reproductive centers in Thailand. It is advisable to choose a center with an NGS platform and a genetic counseling team.

Risk Reminder: PGT technology has inherent limitations, including misdiagnosis due to mosaicism, technical testing errors, and inability to cover polygenic diseases. All PGT results should be interpreted under the guidance of a genetic counselor or reproductive doctor. After pregnancy, formal prenatal diagnosis (amniocentesis or chorionic villus sampling) is still recommended to confirm the fetal chromosome and gene status. This content is for medical science reference only. Please consult a formal reproductive center for specific diagnosis and treatment plans.

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